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Post-Doc Position in Cellular and Molecular Mechanisms of Stroke/Traumatic Brain Injury

Employer
University of Wisconsin- Madison
Location
Madison, Wisconsin
Salary
NIH Stipend Rate
Closing date
May 19, 2023

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This position is in the laboratory of Dr. Raghu Vemuganti, Professor and Vice-Chair for Basic Research, Dept. of Neurological Surgery, Univ. of Wisconsin. The lab is supported by several federally funded (NIH and VA) grants.

 

The primary project is to investigate the role of noncoding RNAs (microRNAs, lncRNAs, and circRNAs), epigenetics, and epitranscriptomics in secondary brain damage. Proficiency in rodent stroke/TBI models, motor and cognitive function analysis, molecular and cellular techniques, including histopathology, immunohistochemistry, Western blotting, PCR, and siRNA work, is preferred. Skills in data analysis, statistics, bioinformatics, and writing are helpful.

 

PhD in neuroscience or biomedical sciences and proficiency in English is required. The incumbent is expected to interact with several scientists, post-docs, grad students, and undergrad students working in Vemuganti Lab.

 

If interested, submit a CV, a brief cover letter indicating prior research experience, career goals, and names of 2 referees to Dr. Raghu Vemuganti (vemuganti@neurosurgery.wisc.edu).

 

 

The University of Wisconsin (Madison, WI, USA) is one of the Big 10 universities with >1,000 active biomedical research laboratories. Madison is one of the best places to live in the United States. The University of Wisconsin is an equal-opportunity employer and encourages applications from underrepresented groups and women.

 

Representative publications from the lab:

 

1. Chokkalla AK, Jeong S, Subramanian S, Vemuganti R (2023). Immunomodulatory role of glycoRNAs in the brain. Journal of Cerebral Blood Flow & Metabolism 43:499-504.

 

2. Mehta SL, Chokkalla AK, Bathula S, Arruri V, Chelluboina B, Vemuganti R (2022). CDR1as regulates α-synuclein-mediated ischemic brain damage by controlling miR-7 availability. Molecular Therapy—Nucleic Acids 31:57-67.

 

3. Davis CK, Bathula S, Hsu M, Morris-Blanco KC, Chokkalla AK, Jeong S, Choi J, Subramanian S, Park JS, Fabry Z, Vemuganti R (2022). An antioxidant and anti-ER stress combo therapy decreases inflammation, secondary brain damage and promotes neurological recovery following traumatic brain injury in mice. Journal of Neuroscience 42:6810–21.

 

4. Morris-Blanco KC, Chokkalla AK, Arruri V, Jeong S, Probelsky SM, Vemuganti R (2022). Epigenetic mechanisms and potential therapeutic targets in stroke. Journal of Cerebral Blood Flow & Metabolism 42:2000-2016.

 

5. Chokkalla AK, Jeong S, Mehta SL, Davis CK, Morris-Blanco KC, Bathula S, Qureshi SS, Vemuganti R (2023). Cerebroprotective Role of N6-Methyladenosine Demethylase FTO (Fat Mass and Obesity-Associated Protein) After Experimental Stroke. Stroke. 54(1):245-254.

 

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