Postdoctoral Fellow

Employer
UC Davis Internal Medicine : Division of Pulmonary, Critical Care & Sleep Medicine
Location
Sacramento, California
Salary
Commensurate With Experience.
Posted
November 16 2020
Position Type
Full Time
Organization Type
Academia

Postdoctoral Fellow

Department of Internal Medicine: Division of Pulmonary, Critical Care & Sleep Medicine

We seek postdoctoral candidate with an interest in pulmonary vascular signaling, cell biology, and cell metabolism to study the molecular and cellular mechanisms of pulmonary vascular remodeling as it relates to pulmonary arterial hypertension (PAH) with specific focus on growth suppressor proteins and cellular metabolism. The successful applicant should have expertise in cellular and molecular biology techniques (i.e. western blotting, cell transfections/ infections, proliferation and apoptosis assays). Expertise in cellular metabolism, cell culture techniques and small animal (rats and mice) models of pulmonary hypertension is appreciated.

To be considered for the position, please send in a detailed CV with methodological expertise, a list of scientific publications, and the contact details of three referees to massad@ucdavis.edu. Salary will be according to NIH scale and commensurate with experience. Position available immediately.

Goncharova lab research interests have focused on the molecular and cellular mechanisms of pulmonary arterial hypertension (PAH). We use molecular and cellular techniques, human tissues and cells, transgenic mice, and rodent models of pulmonary hypertension to dissect new molecular signaling hubs driving hyper-proliferation of resident pulmonary vascular cells, pulmonary vascular remodeling and PAH and to identify and pre-clinically validate novel molecular target pathways for therapeutic intervention. Ongoing projects are focused on the role of mTOR and HIPPO signaling networks in pulmonary vascular cell remodeling and right ventricular dysfunction in pulmonary arterial hypertension; the role of GATA6 transcriptional factor as a regulator of pulmonary vascular cell-cell communications, BMP signaling and inflammation in PAH; and role of lipid metabolism in pulmonary vascular remodeling.

UC Davis commits to inclusion excellence by advancing equity, diversity, and inclusion in all that we do. We are an Affirmative Action/Equal Opportunity employer, and particularly encourage applications from members of historically underrepresented racial/ethnic groups, women, individuals with disabilities, veterans, LGBTQ community members, and others who demonstrate the ability to help us achieve our vision of a diverse and inclusive community. For the complete University of California nondiscrimination and affirmative action, policy see: http://policy.ucop.edu/doc/4000376/NondiscrimAffirmAct. If you need accommodation due to a disability, please contact the recruiting department. Under Federal law, the University of California may employ only individuals who are legally able to work in the United States as established by providing documents as specified in the Immigration Reform and Control Act of 1986. Certain positions funded by federal contracts or sub-contracts require the selected candidate to pass an EVerify check. More information is available http://www.uscis.gov/e-verify. UC Davis is a smoke & tobacco-free campus (http://breathefree.ucdavis.edu)

THIS IS NOT AN H1- B OPPORTUNITY.

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