Postdoctoral position in Neurobiology, Genetics and Ocular Diseases at UCSF

Location
University of California, San Francisco (UCSF)
Salary
Competitive salary with living allowance
Posted
January 22 2019
Position Type
Full Time
Organization Type
Academia

A postdoctoral position is immediately available in the laboratory of Sai Nair, PhD at the University of California, San Francisco. We are looking for a highly motivated candidate with interest in understanding the mechanisms underlying neurodegenerative diseases. Our lab studies the genetics of ocular diseases with a primary focus on glaucoma and other retinal disorders. Our goal is to identify genes, molecular mechanisms and cellular networks contributing to ocular neurodegenerative diseases. The lab employs a variety of multi-disciplinary approaches to dissect the role of complex disease associated genes at a mechanistic level.  We employ human genetics-based approaches to identify candidate genes and test their importance in disease pathogenesis using mouse genetic models.

 The successful candidate will work in a collegial, interdisciplinary research environment and integrate the use of animal models human genetics, cell-biology, functional genomics and physiology based experiments. We provide competitive salary and an additional housing allowance. UCSF provides quality on-campus housing and related services to postdocs. More information on our research can be found at http://ophthalmology.ucsf.edu/nairlab/

 The successful applicant will be part of a structured departmental training program to prepare the candidate for a career in science. Interested candidates should e-mail a cover letter, the names and contact information of two to three referees to Saidas Nair (saidas.nair@ucsf.edu).

Relevant Publications

  1. Choquet H et al. A multiethnic genome-wide association study of primary open-angle glaucoma identifies novel risk loci. Nat Communications. 2018 Jun 11;9(1):2278.
  2. Paylakhi SH et al. Müller glia-derived PRSS56 is required to sustain ocular axial growth and prevent refractive error. PLOS Genet. 2018; 14(3):e1007244.

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